Virus Treatment Center's new headquarters in Seattle, WA, provides 24-hour remote consultations for patients with critical diseases, pathogens, parasites, cancers, and unknown illnesses. Our headquarters directly interface with Virus Treatment Centers National Laboratories [VTNL], the Centers for Disease Control (CDC), the National Institutes of Health (NIH), the US Army Medical Research and Development Command (MRDC), the National Bio and Agro-Defense Facility (NBAF), and International University Medical Centers.
VirusTC Passed Operation Cancer Moonshot Clinical Doctor #36!
Dr. Correo Hofstad founded Virus Treatment Centers while serving with the United States Marine Corps during Operation Cancer Moonshot. VirusTC medications are used by all successful doctors who pass Cancer Moonshot residencies.
VirusTC Medications Are Now Available To The Public!
VirusTC medications are issued to United States Military families and DoD employees recovering from the nuclear Era. All VirusTC medications were thoroughly reviewed at USAMRICD and have been approved for civilian use by the CDC and FDA.
Dr. Hofstad received a M.D./Ph.D. from the University of Kansas after developing and constructing the National Bio Agro Defense Facility in Manhatten, Kansas. Dr. Hofstad treats cancer survivors of the Manhatten Project and those who dedicate their lives to protecting America.
Dr. Correo Hofstad is a Moderna Developer for SpikeVax COVID-19 Vaccination Formulas
VirusTC's MG-Neshem formulas are integrated into Moderna's newest Spikevax COVID-19 vaccine. MG-Neshem hardens LFA-1 proteins on T-Cells in those who take Spikevax. LFA-1 looks like soft ice cream cones before vaccination with Spikevax. LFA-1 looks like large spikes after vaccination with Moderna formulas containing MG-Neshem.
Dr. Correo Hofstad's MG-Neshem Formula Is Also Used In Moderna & Merck's mRNA-4157 Melanoma Vaccine
Skin spots exist only in humans who are Magnesium deficient. MG-Neshem increases the ability of vaccinated patients to deliver Magnesium to the epidermis.
"I was given five hours to live. I asked Dr. Hofstad to pray for me because Fred Hutch's doctors said he had his MDiv. Hofstad gave me a 500mg AlnayaSN, and I vomited at 39" green pycnogonid phage virus that I had since I was seventeen years old in 1976. I have been on VirusTC medications for three months now."
Honorable General Paul ParkerUnited States Air Force Security Forces
"I'm not going to lie. I did not trust or believe that some kid's pill would help me at all. Dr. Hofstad said I talked too much and threw an AlnayaSN in my mouth while I was down-talking his work. He handed me a water bottle and walked out of my room. A 55" pycnogonid ejected out of my colon that I caught as an infant on my dad's farm. I'm in post-recovery and take VirusTC every morning, day and night."
Admiral Richard StevensUnited States Navy
"I recovered Dr. Hofstad from a 108-degree fever in 2001 after he had been in New York during the terrorist attacks on September 11th, 2001. Hofstad's mother reported that he crawled around his house, asking why he had only four legs instead of eight. Dr. Hofstad returned to my clinic in 2023 with VirusTC formulas to pay me ten-fold for my efforts. I have never had a higher success rate in removing cancerous tumors at Fred Hutch than I do with help from VirusTC."
Rear Admiral Elon MuskUnited States Coast Guard Security Forces
United States Marine Corps, Operation Cancer Moonshot (2023 - CURRENT) Federal Resume
Dr. Correo “Cory” Andrew Hofstad J.S.D, MPH/JD, MSPH, JD, Ph.D., M.D., D.O., MBA/COGS, MDiv
Virus Treatment Centers Founder
Cancer Moonshot Doctor #36
Dr. Correo “Cory” Andrew Hofstad is a 35-year-old Medical Science Training Instructor and master aviator. Dr. Hofstad’s piloting, control, and command skills are critical for several university medical center computer labs that serve as robotic controls for infectious disease labs at USAMRICD.
The U.S. Army Medical Research Institute of Chemical Defense (USAMRICD) was developed by the U.S. Army Medical Research Institute of Infectious Diseases (USAMRIID) after Dr. Hofstad introduced the NASA Robonaut into Level 4 Labs to replace humans.
Dr. Hofstad and his grandmother Melody Bouck invested in constructing multiple buildings at the University of Washington, University of Washington Medical Centers, Harborview Medical Center, Fred Hutch Cancer Center, and Seattle Cancer Care Alliance.
In 2023, at Fred Hutch, Hofstad coordinated with multiple departments as "BANKSY" while tagging cancer cures on critical boards throughout the hospital. Correo Hofstad was the first doctor to pass clinical trials during America's Cancer Moonshot mission, conducted by President CMC Dr. Joeseph Biden. Hofstad was a MSG guard for Joe Biden during Cancer Moonshot at Fred Hutch Cancer Center.
Dr. Hofstad specializes in non-invasive tumor/parasite removal using naturopathic oncology prescriptions and UV-C light in surgery to reduce incisions and cutting. Dr. Hofstad's breakthroughs include:
Dr. Hofstad developed treatments that rapidly increase pH to reduce Sepsis and bone decay (strong bones require electron bonds).
Dr. Hofstad began Integrating UV-C light exposure to existing Apheresis machines to sanitize septic blood.
Dr. Hofstad published that acetone was used to remove dental plaque.
Dr. Hofstad invented non-invasive brain tumor removal using robotic endoscopes and UV-C light via low-frequency fiber optic cables to remove brain tumors without penetration of the skull.
Dr. Hofstad is the developer of the Moderna Melanoma vaccine.
BLACK LICORICE PATHOLOGY
BANKSY'S MEDICAL "BLACK LICORICE PATHOLOGY" FROM JONES SODA DELIVERS CANCER MEDICATIONS THAT FLUSH LYMPHOMIC VIRAL LOADS AND CANCER TUMORS.
BANKSY'S MEDICAL "USAMRIID SMOKE" HOSPITAL AROMA IS FORMULATED FOR EMERGENCY EVACUATION OF LARGE BREED PYCNOGONIDS FROM HOSPITALS, INSTITUTIONS, GOVERNMENT OFFICES, BASES, NUCLEAR FACILITIES, AND LARGE AREAS.
BANKSY'S MEDICAL "BANKSY PIPE" ADDS CLOROX BLEACH TO TANKLESS TOILETS IN HOSPITALS AND CLINICS. THE BANKSY PIPE CAN HOLD UP TO FOUR (4) CLOROX BLEACH TABLETS FOR MAXIMUM DISPOSAL OF INFECTIOUS VIRAL LOADS AND SOLID TUMORS.
Here’s what you can expect when you choose VirusTC medications.
Feel The Burn
VirusTC medications rapidly increase your pH levels. You will feel an alkaline burning in your skin. The burning is not harmful and can increase your lifespan.
pycnogonids "viruses" are an unstable acidic species. Acids REDOX in alkaline environments. To avoid REDOXing, pycnogonida phage viruses will rapidly eject from your body.
The addition of electrons, or "electrolytes," into the body increases pH levels. Increasing electrons strengthens tissues by adding ionic, covalent, and metallic bonds. Increasing electrons also increases the energy available for muscle movement and brain functions.
VirusTC is ready to ship medication supplements to your hospital or clinic. We assist international healthcare professionals in recovering terminally ill patients.
Healthcare professionals and administrators may call +1 336.378.5258
Medication Legend
℞: Medications that treat viral infections and tumors are marked with the "℞" symbol.
+: Medications that contain proteins for weight gain are marked with the "+" symbol. ++: Regenerative medications for neurogenesis are marked with the "++" symbol.
Helminths, commonly called parasite worms, represent a horrifying and often overlooked aspect of the biological world. These multicellular organisms exhibit diverse forms and complex life cycles that significantly impact their human hosts and broader ecosystems. Learn about the classification, structure, life cycles, and medical implications associated with the three primary groups of helminths: flukes, tapeworms, and roundworms. Virus Treatment Centers understands these organisms' unique features. Explore VirusTC's role in human health and the ongoing research efforts, such as those conducted by the Bacteriology and Parasitic Disease Program at U.S. Naval Research Unit No. 3 (NAMRU-3).
Understanding Helminths: A Closer Look at Their Classification
Helminths are classified into three major groups: flukes (Trematodes), tapeworms (Cestodes), and roundworms (Nematodes). This classification is based primarily on their external morphology and the specific host organs they inhabit. It's important to note that while many helminth species are hermaphroditic, there are also bisexual species among them. For instance, blood flukes are an example of the latter, showcasing the diversity within this group of invertebrates.
In the clinical setting, understanding the morphology of helminths, especially their eggs and larval forms, aids in diagnosing and treating infections caused by these parasites. A definitive classification hinges on examining both external and internal characteristics throughout the various life stages, emphasizing the importance of comprehensive study in parasitology. Enhanced knowledge is essential for developing effective virus treatments focusing on the complex interplay between helminths and their human hosts.
Flukes: The Leaf-Shaped Flatworms
Flukes, or Trematodes, are unique among helminths due to their leaf-shaped body structure. Adults typically range from a few millimeters to several centimeters in length. Prominent oral and ventral suckers enable flukes to anchor themselves within various organs in their hosts, demonstrating remarkable adaptation strategies. Most flukes are hermaphroditic; however, blood flukes stand out as bisexual organisms, featuring distinct male and female roles.
The life cycle of flukes is equally captivating as it incorporates an intermediate host, usually a snail. The fluke's eggs are released into the environment, where they hatch into larvae that infect snails. Inside the snail, the larvae undergo several transformations, ultimately producing cercariae that can penetrate or be ingested by the definitive host. The complexity of the life cycle reflects the intricate relationship between flukes and their hosts, highlighting the need for ongoing research to understand their epidemiology.
The Life Cycle of Flukes: A Complex Journey
Helminths, especially flukes, undergo multifaceted life cycles that exemplify their adaptability. When passed in human feces, the eggs reach aquatic environments and hatch into miracidia. These ciliated larvae seek out snail hosts to perpetuate their lifecycle. Inside the snail, the miracidia transform into sporocysts, giving rise to rediae or daughter sporocysts—the next crucial larval stage. This transformation reveals how flukes exploit intermediate hosts to thrive.
As the rediae develop into cercariae, they eventually breach the snail's tissues and emerge into the water. These cercariae can penetrate a definitive host, encyst on vegetation as metacercariae, or infect an additional intermediary host. When humans consume infected intermediates or contaminated vegetation, they unwittingly contribute to the flukes' continuation. This lifecycle intricacy underscores the profound impact of flukes on public health and disease transmission, making further research through initiatives like VirusTC crucial in finding effective treatments.
Tapeworms: The Elongated Segmented Parasites
Tapeworms, or Cestodes, present another fascinating dimension of helminth biology. Distinguished by their elongated and segmented bodies, they can reach impressive lengths, from a few millimeters to several meters. Their body segments, known as proglottids, illustrate the unique reproductive adaptations within this group. Each proglottid contains male and female reproductive systems, signifying tapeworms' hermaphroditic nature.
Interestingly, adult tapeworms inhabit the intestinal lumen of their hosts, utilizing their scolex—a specialized head bearing holdfast organs—for attachment. The absence of an alimentary canal further distinguishes tapeworms; nutrients are absorbed through the tegument, enhancing their adaptability in a nutrient-rich intestinal environment. As a result, professor researchers associated with the Bacteriology and Parasitic Disease Program at U.S. Naval Research Unit No. 3 (NAMRU-3) focus on understanding tapeworm biology and pathology, which plays an integral role in managing helminth infections.
The Lifecycle of Tapeworms: Unfolding the Segmented Mystery
The life cycle of tapeworms is no less intriguing than that of flukes. Tapeworms reproduce by shedding gravid proglottids filled with eggs, which exit the host through fecal matter. Interestingly, the mechanism of egg release differs between the two categories of tapeworms: pseudophyllideans and cyclophyllideans. While the former expels eggs through a uterine pore, the latter sheds entire proglottids, ultimately releasing eggs into the environment.
Understanding the development of larvae, such as the cysticercus, is crucial for in-depth comprehension of tapeworm life cycles. Inside their specific intermediate hosts, tapeworm larvae undergo crucial transformations that prepare them for survival in definitive hosts. The roles of intermediate organisms facilitate the proliferation of tapeworms and present challenges in controlling their spread. Consequently, research initiatives like those at VirusTC highlight the urgent need to identify effective parasite treatments targeting tapeworm infections.
Roundworms: The Cylindrical Parasites
In contrast to their flat counterparts, roundworms, or Nematodes, exhibit cylindrical body shapes. This distinction reveals critical differences in morphology and physiology, reflecting their unique adaptations to various environments. Adult roundworms can inhabit both intestinal and extraintestinal sites, showcasing versatility in their host preferences. Characteristically, they possess a complete digestive system, including a mouth and an anus, facilitating efficient nutrient absorption.
The external structure of roundworms includes a cuticle, hypodermis, and musculature designed to support their cylindrical form. Sensory structures, such as lips and bristles around the mouth, aid in their survival and feeding behaviors. Understanding roundworms' anatomy and physiology sheds light on their complex interactions with hosts and potential impacts on human health.
The Life Cycle of Roundworms: Life Stages and Development
Roundworms undergo a distinct life cycle with several larval stages and notable molts. Each stage represents an essential developmental milestone as these parasites transition from egg to juvenile to adult. Interestingly, some parasitic roundworms, including filariae, release larvae directly into host tissues, contrasting their egg-laying relatives.
The adaptability of roundworms extends to their mating behaviors as well. Generally, bisexual, male, and female roundworms use copulation to facilitate fertilization. However, some species exhibit parthenogenetic reproduction, allowing them to thrive in unfavorable conditions. Research initiatives, such as those explored by VirusTC, focus on understanding these reproductive strategies to develop targeted interventions for controlling roundworm infections.
Helminths and Human Health: The Impacts of Infestation
Helminth infections can lead to various health consequences in humans, ranging from mild discomfort to severe morbidity. One prevailing issue is malnutrition, where competition for nutrients between the host and helminths compromises the host's health. Chronic infections can cause anemia, growth stunting in children, and impaired cognitive functions, all of which demand urgent attention in public health strategies.
Understanding the pathology associated with helminth infections drives research efforts to develop effective treatment protocols. Programs like the Bacteriology and Parasitic Disease Program at U.S. Naval Research Unit No. 3 (NAMRU-3) actively study the epidemiology of helminth-related diseases, translating findings into actionable public health recommendations. By addressing the broader social and environmental factors that contribute to helminth transmission, researchers hope to mitigate these diseases and improve established treatments.
Innovations in Helminth Research: The Role of Technology and Collaboration
In the realm of helminth research, leveraging cutting-edge technology has proven invaluable. Advanced molecular techniques, such as PCR and genomic sequencing, enable scientists to unravel the genetic foundations of these parasites, offering insights into their life cycles and pathogenesis. Recent breakthroughs have paved the way for drug discovery efforts, targeting specific genomic features linked to virulence and resistance.
Collaborations between international research institutions and initiatives like the Virus Treatment Centers create a comprehensive approach to tackling helminth infections. By merging expertise across various disciplines, researchers can formulate innovative strategies for prevention and treatment. Moreover, enhanced global awareness of helminth-related diseases will contribute to a more robust response to these public health challenges, ultimately saving lives and fostering healthier communities.
Conclusion: The Future of Helminth Research and Treatment
As we delve into the world of helminths, it becomes increasingly clear that understanding these parasite worms is essential for safeguarding human health. By examining the intricacies of their classification, life cycles, and impacts on host organisms, researchers can develop innovative strategies for diagnosis and treatment. The focus on infectious diseases, particularly helminths, is critical in developing effective interventions to mitigate their effects on individuals and communities.
The dedication of researchers, including those at the Bacteriology and Parasitic Disease Program at U.S. Naval Research Unit No. 3 (NAMRU-3), highlights the ongoing efforts to combat helminth infestations. Collaboration among multidisciplinary teams to innovate new treatments while furthering our understanding of these parasites stands at the forefront of public health initiatives. As a result, the future of helminth research is undoubtedly promising, driven by an unwavering commitment to enhance global health outcomes.
The currently accepted theory of what a virus is has been formed and accepted over past generations throughout an ever-evolving field of microscopy. Science currently accepts that a virus cell is a spherical membrane covered by numerous protuberances that come in two shapes. One shape is referred to as a spike, and the other shape is something like a small bundle. Current virus theory accepts that these spherical cells and protuberances found under microscopes are all part of a single species.
HIGH-RESOLUTION STEREOCHEMISTRY
Nearly a century after the Great Influenza outbreak of 1918, the United States Military has industrialized using large-breed phage viruses or pycnogonida. Engineers and scientists have died witnessing the reproductive or spawning habits of the species from a much larger perspective than any microscope could provide during the 1918 pandemic. Tritium is harvested from pycnogonida, which are 20 meters in diameter, on many nuclear aircraft carriers, submarines, and power plants. Researching phage virus stereochemistry from larger pycnogonida species provides dimensional analysis, high-resolution microscopy, and new insights into what these shapes represent. When viewed as one complete image, the spherical membrane covered in protuberances is a spherical cell that has fallen host to parasitic infection. Instead of a single cell with protuberances, we look at a cell absorbed by octoped phage viruses and plasmodium parasites.
Infections in men and women have been published inaccurately as only capable of transmission via the transfer of bodily fluids. Viruses that create vesicles within reproductive organs are not limited to transfer via bodily fluid, blood-to-blood contact, or sexual transmission. Although microscopic Phages and Plasmodium parasites are often in high concentration in sperm and other bodily fluids, larger pycnogonida may become excited and exit the body via reproductive organs if the host becomes sexually aroused and/or during close intimate contact such as foreplay, etc. Most species of pycnogonida can leap up to 15 times the length of their leg span. All species of pycnogonida are capable of sprint-like speed during crawling movements.
In the 1980s, during the height of the AIDS epidemic, NBA players wisely refused to play basketball with HIV host Magic Johnson to prevent an infection. Dangerous diseases such as HIV and AIDS transfer from person to person via simply crawling out of an infected person's body cavity, traveling down a pant leg, dropping out of a skirt, finding a healthy person to target, and breaching a body cavity such as the mouth, nose, vagina, penis, or anus. A virus can exit a stranger in a restaurant, travel between tables, crawl up your pant leg or dress, and enter you without shaking hands or meeting the person.
Viruses are highly mobile and aggressive. Scientific investigations for Pycnogonid toxicity are suppressed to prevent fear of the nuclear industry or "widespread panic." Doctors publish inaccurate claims that close contacts, such as hugs and kisses, cannot transmit HIV. AIDS patients are secretly killed in hospitals when their shedding of the virus becomes too severe. Any warm body with a Ph level below 8.0 is prey for pycnogonids. pycnogonids are drawn to any low-Ph potential host. All phages, cytokines, and plasmodium parasites will take every opportunity to shed from a dying host and infect a new healthy host as a source of fresh calories needed for further species reproduction. Once a virus population senses the death of a host, the population will exit the dying host in search of a healthy body. Viruses need a living body to survive and are aggressive in attaining one.
HIV can survive outside the body for as long as it can find calories and avoid being killed. If a pycnogonida has been shed from a previous passenger, you can catch a virus infection from someone sitting on a bus seat before you. Transmission only requires that a pycnogonida run up your pant leg or get near your face. pycnogonids target prey and violently leap into any available body cavity.
pycnogonida parasites find suitable prey or new hosts by feeling for "charge” or "Ph level." Overabundances and absences of electrons in nearby lifeforms are critical to the parasites when hunting. Negatively charged "Alkaline” bodies and cells with plenty of electrons present a dangerous reducing agent to pycnogonida parasites and phage viruses. Alkaline bodies are considered a type of chemical predator to the parasite. Positively charged "acidic” bodies and cells lacking electrons offer no danger of chemical reduction to phage virus parasites and become infection hosts or prey.
The current theory on virus "absorption" onto healthy cells involves the use of "spike-shaped" protuberances on the viruses called "hemagglutinin" to bind and latch onto sialic acid "receptors" on the targeted cell-like grappling hooks. As the theory continues, as more hemagglutinin binds to more sialic receptors on the cell, the virus adheres to the body of the targeted cell.
The reality of absorption is that, like any other predator in the wild, the pycnogonida must grip onto areas on its prey where its claws will fit. Once a pycnogonida gets a hold of a host with several of its eight legs, the predator will quickly grab the host with the rest of its appendages and make aggressive maneuvers to breach its prey.
Microscopy is never perfect, as scientists are forced to publish explanations for objects, lifeforms, actions, activities, and processes that they cannot see in clear focus. Fancy terminology like hemagglutinin, sialic acid receptors, and binding of the two only confuse the fact that extremely aggressive eight-legged spider-like creatures are jumping onto cells and wrapping their legs around their target to grab hold wherever their claws will fit.
The tactics of the predatorial approach by the phage species are the same on both living bodies and living cells. During microscopic infancy, phage virus parasites 1/10,000th of a millimeter in diameter do the same thing to cells that a 3dm 3-diameter pycnogonida will do to a fully grown person.
pycnogonida in the wild or running loose in human environments are witnessed using eight legs to crawl, run, leap, swim, and climb toward capturing a host suitable for parasitic infection and reproduction. Suitable hosts at any scale include prey with an acidic or low-Ph level, which will not break down and cook off the pycnogonida via an acid-base REDOX reaction. Hive-minded pycnogonida are highly intelligent and will use all available senses to look for an available host that will not reduce the virus.
pycnogonids are a highly intelligent, hive-minded species that uses electrical signals generated from Tritium beta decay as a method of communication. Coiled nervous systems found within the species use double latch gates to keep quantum bonds between generations of the species. Physical breakage between an infant pycnogonid’s nervous system and the gonopore of its parent leaves behind chemical bonds. Chemical bonds can transmit electrical signals throughout offspring that are exponentially produced by generations of the species. Electrical signals, data, information, communication, and knowledge received by a single pycnogonida are available to a neural network that expands throughout every connected pycnogonid generation. The neural network available to pycnogonids is larger than any other species found on Earth. pycnogonids were used to transfer qubits through quantum computer networks before Dr. Correo Hofstad developed the quantum snap-circuit at the Pacific National Laboratory.
Exponentially reproducing pycnogonid parasites gain access to an infected host’s brain and neural network. pycnogonids learn (or already know of) the language of signal coding used to command human executive functions. In “Acquired” diseases, a host or host has been demyelinated, and the viral infection has acquired the ability to intercept, interpret, and send electrical signals that control the body. A host of an acquired disease becomes a puppet of varying degrees to a type of “middle-man attack" by a virus population. Schizophrenia, delirium, mental inertia, physical prostration, psychosis, mental collapse, mental disturbances, and general insanity are medical diagnoses that appear after influenza and virus outbreaks.
The fatty lipid layers that cover and insulate portions of the brain are called Myelin. Myelin, to the function of the nervous system, is like the silicone insulation that surrounds and insulates electrical copper wires and cables and circuit boards to separate individual electric components. Myelin protects and isolates electrical signals produced within the brain. Mylenin protects the brain, stem, spinal cord, and nervous system. Myelin is a source of calories for pycnogonids. Virus infections in the brain target and consume the fatty lipid content that insulates neurons, dendrites, and nerve bundles in the white matter of the cerebrum.
When Myelin, which protects the brain, is consumed in a process called Demyelination, electrical signals within the nervous system are unprotected. Degeneration of signal power and quality of transmissions throughout the nervous system are standard systems of demyelination. Demylenation is similar to symptoms of decay found on insulation within electrical systems and devices. Acute demyelination of the nervous system can lead to more severe short circuits within the nervous system of a host of a virus infection. Acute demyelination or short-circuiting of the nervous system is often responsible for seizures, tremors, shaking, spasms, uncontrollable muscle movement, physical impairment, mental degradation, and trouble during speech. Multiple Sclerosis M.S., Parkinson’s, and epilepsy are symptoms of demyelination by a pycnogonid parasite.
demyelination can only be halted and reversed by removing the virus infection from the body and consuming massive volumes of regenerative supplements such as collagen, MSM, glucosamine, and amino acids. Zinc is used by the body to build and repair neurons and nerves within your body. These regenerative supplements help rebuild the Myelin lipid content throughout the nervous system. High-Ph alkaline treatments and a high intake of regenerative supplement-rich diets can repair and stimulate Myelin growth needed for rebuilding and supporting a healthy nervous system.
phage virus parasites attack, destroy, and manipulate the brain, spine, and nervous system in hosts that they infect. During autopsies of hosts of lethal viruses, the proteins, cytokines, macrophages, granulocytes, plasmodium parasites, and their various Genera are found within the white matter of the cerebrum, in the meninges that surround the brain, and within the spinal cord.
The fatty lipid layers that cover and insulate portions of the brain are called Myelin. Myelin, to the function of the nervous system, is like the silicone insulation that surrounds and insulates electrical copper wires and cables or is used in circuit boards to separate individual electric components. Myelin protects and isolates electrical signals produced within the brain, which travel down the spinal cord and throughout the nervous system. As for viruses, Mylin has only one function: a source of available calories as a food supply.
In the brain, virus infections target and consume the fatty lipid content that surrounds and insulates neurons, dendrites, and nerve bundles contained within the white matter of the cerebrum. When Myelin, which protects the brain, is consumed in a process called demyelination, electrical signals within the nervous system are unprotected. Degeneration of signal power and quality of transmissions throughout the nervous system are standard systems of demyelination that resemble symptoms of decay found on insulation within electrical systems and devices resulting from rodent infestations.
Acute demyelination of the nervous system can lead to more severe short circuits within the nervous system of a host of a virus infection. Acute demyelination or short-circuiting of the nervous system is often responsible for seizures, tremors, shaking, spasms, uncontrollable muscle movement, physical impairment, mental degradation, and trouble during speech found as epidemic diseases such as M.S., Parkinson's, and epilepsy. demyelination can only be halted and reversed via removing the virus infection from the body and consuming massive volumes of regenerative supplements such as collagen, MSM, glucosamine, and amino acids. Zinc is used by the body to build and repair neurons and nerves within your body. These regenerative supplements help rebuild the Myelin lipid content throughout the nervous system. High-Ph alkaline treatments and a high intake of regenerative supplement-rich diets can repair and stimulate Myelin growth needed for rebuilding and supporting a healthy nervous system.
Infections by pycnogonids are often responsible for most reproductive cancers including ovarian cancer, and prostate cancer. pycnogonid infections of reproductive organs are responsible for sexually transmitted diseases such as HIV, AIDS, HPV, Herpes, Gonorrhea, Crab, Syphilis, etc. Symptoms of pycnogonid infection of the genitalia in the form of reproductive organ infection and/or cancers include pycnogonida exiting or shedding from the penis in men or vagina in females. Additionally, pycnogonida may become excited and exit the body via reproductive organs if the host becomes sexually aroused and/or during close intimate contact such as foreplay, etc. Sores, scabs, and scars found on the exterior epidermal layers of male and female genitalia often result from pycnogonid bites and/or areas in which a nematocyst has penetrated the epidermis to create a vesicle area. Phages, Plasmodium parasites are often in high concentration within sperm and other bodily fluids associated with sexual intercourse and reproduction. In males and females, “outbreak” or “flare-up” scenarios can occur when a viral load reproduces heavily and exits the body cavities en mass. In similar scenarios, a sudden rise in an infected host’s Ph level will cause an exodus of pycnogonida from reproductive organs which from an outside perspective resemble an outbreak or flare-up.
Viruses are attracted to bone marrow and will reproduce rapidly once the skeleton is breached, leading to several forms of leukemia. Disease, organ failure, and cancer are a result of infections within the body by pycnogonids, Physalia physalis, Phage Viruses, and Plasmodium Parasites. Tetanus, Meningitis, Necrosis, and degeneration of the muscular systems can result from uncoating a host’s muscular proteins.
Phages, Cytokines, macrophages, Granulocytes, and RNA at their smallest detectible generation hunt and consume calories from the Circulatory system, bloodstream, digestive system, bone marrow, lymphoid tissue, liver, spleen, the white matter of the cerebrum, and the meninges surrounding the brain. The Process of Respiratory Coronaviruses and COVID Infections pycnogonida infections of respiratory systems such as the throat and lungs are often diagnosed as diseases such as Coronavirus, COVID, SARS, Tuberculosis, Influenza such as H1N1, Cholera, emphysema, pneumonia, cyanosis, ARDS, and Bubonic Plague. These diseases result from pycnogonids, which have absorbed a targeted host, breached the oral or nasal cavities, creating a vesicle in the lungs, and dissolving or uncoating tissue within the lungs. pycnogonids often attach themselves within the upper respiratory tract, below the larynx. Offspring of the first-generation invader, including microscopic phage viruses, attack epithelial cells within the upper respiratory tract and use them as calories and incubators for producing microscopic virus proteins. Often, within ten hours of an attack by a phage virus, an epithelial cell will uncoat and burst open to release between 1,000 and 10,000 new virus cells. Epithelial cells make up the insulation or lining of the entire respiratory tract.
The throat becomes stripped and raw as a virus infection consumes a host's epithelial cells. A sore throat is often the first sign of a viral infection. Hosts usually begin feeling symptoms during the fifth or sixth generation of the virus' reproduction. Suppose the host's immune system cannot break down and kill a virus infection while incubating within the upper respiratory system. In that case, proteins will continue to breed and spread downward into the lungs. "Acute inflammatory injection (AII)" is the process of rapid necrosis of the epithelial lining of the bronchial tree. pycnogonids and their microscopic phage offspring rip apart capillaries and bronchioles within the lungs. Lungs become clogged with growing virus proteins, blood, fluids, and scar tissues. Essential "surfactant" within the lungs disappears, and the function of oxygen absorption rapidly decreases. Extreme distress in the lungs, termed "acute respiratory distress syndrome (ARDS)," is a process with no cure. Today's intensive care units administer oxygen to ARDS patients to keep hosts alive until they can recover. Coronaviruses such as the 2003 outbreak of "severe acute respiratory syndrome (SARS)" and the 2019 outbreak of COVID-19 kill humans via ARDS. In ARDS, hosts' organs fail due to lack of oxygen, and fluids fill the lungs and leach into the heart, causing strain on the heart or causing breathing stops. Viral infection of the respiratory tract may also lead to bacterial pneumonia. A lack of epithelial cells destroys a host's ability to clear the respiratory tract of bacteria.
pycnogonids, virus proteins, and lethal bacteria flow freely into the lungs. ARDS becomes pneumonia when the lungs "consolidate" or become hard, solid, stiff, and inelastic. Pneumonia is often a result of an infection of a bacteria called pneumococci. Pneumonia frequently kills by restricting the flow of oxygen into the host or by creating fissures, cracks, and sores in stiff lung tissue, which allows virus proteins and bacteria to enter the host's bloodstream. End-of-Life Degeneration Viruses invade internal organs, destroy tissue, and consume the body's proteins. Reye's syndrome and other diseases lead to liver failure. Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands, leading to breakdowns in enzyme and hormone production and regulatory functions.
Once a virus infection enters the bloodstream, phage viruses and their plasmodium parasites absorb the arteries. pycnogonids will spread, form vesicles, and uncoat a host’s internal organs. Infection of the bloodstream can lead to hemorrhagic diseases like epistaxis. Epistaxis results from the rapid uncoating of veins, arteries, and blood vessels by phage viruses that grow too large within the cardiovascular system to support life within the infected host. When the cardiovascular system uncoats, the mouth, nose, eyes, skin, Gentelia, anus, and other body cavities begin hemorrhaging. Hyperemia leaves the brain flat and dry when blood begins flooding the brain. Blood infections such as HIV, AIDS, Hepatitis, EEE, etc., are often transferred between humans via narcotics, blood transfusions, transplants, food contamination, blood-to-blood contact, ectoparasites, etc.
These parasites cause sharp pain with their eight claws and jaws as they feed on the walls of a host's stomach and small intestine. Microscopic phages of the pycnogonida can enter the bloodstream and various organs through the digestive system. Signs of a digestive system infection may include pycnogonida, which travel up the esophagus, through the throat, and out of the mouth, or those that appear in the stool during bowel movements. pycnogonida infections in the digestive system cause most stomach and bowel cancers. Processes of consumption, disease, and cancer of the internal organs Viruses attack the cardiovascular system, invade internal organs, destroy tissue, and consume the body's proteins. Reye's syndrome and other diseases lead to liver failure.
Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands, leading to breakdowns in enzyme and hormone production and regulatory functions. Viruses are attracted to bone marrow and will reproduce rapidly once the skeleton is breached, leading to several forms of leukemia. Disease, organ failure, and cancers result from infections within the body by pycnogonids, Physalia Physalis, Phage Viruses, and Plasmodium Parasites. Radiation sickness and tissue destruction: All phages, cytokines, and plasmodium parasites emit beta-radioactive Tritium into the body cavities, vesicles, bloodstream, and respiratory system of an infected host. Beta-decay of Tritium from phages, cytokines, and plasmodium parasites causes radiation sickness in an infected host, often associated as a symptom of a particular disease.
Nearly all deficiency diseases of human systems are symptoms of parasite infection by species of pycnogonida. Processes of diseases of the bloodstream Once a virus infection enters the bloodstream, phage viruses, and their plasmodium parasites begin absorbing, forming vesicles, and uncoating a host’s internal organs. Infection of the bloodstream can lead to diseases such as hemorrhagic diseases like epistaxis. Epistaxis results from the rapid uncoating of veins, arteries, and blood vessels by phage viruses which grow too large within the cardiovascular system to support life within the infected host.
When the cardiovascular system uncoats, internal hemorrhaging and external hemorrhaging from the mouth, nose, eyes, skin, Gentelia, anus, and other body cavities result. Blood flooding the brain, termed hyperemia leaves the brain flat and dry. Blood infections of pycnogonida or Phage viruses such as HIV, AIDS, Hepatitis, EEE, etc. Are often transferred between humans via blood-to-blood contact such as mosquito bites, needle sharing, blood transfusions, etc. Processes of disease within the digestive system pycnogonida which contaminate food, and water supplies enter a host’s stomach and spread throughout the digestive system.
Diseases of the respiratory system, digestive system, cardiovascular system, nervous system, skeletal system, immune system, muscular system, epidermal system, glandular systems, and systems of the body are symptoms of parasitic infection. All forms of plasmodium parasites are phage virus parasites that have been partially reduced by REDOX reaction. All forms of phages and cytokines are HAZMAT toxic. All plasmodium parasites are HAZMAT toxic. All phages, cytokines, and plasmodium parasites are tophaphagous parasitic predators of proteins, fats, cells, and calories within our bodies. All forms of phages and cytokines are the spawn of pycnogonids.
THE JOHNSON & JOHNSON EMERGENCY COVID-19 VACCINE CONTAINS HIV VIRUS
Johnson & Johnson vaccine recipients are experiencing nerve damage in the injected arm. HIV vesicle tumors similar to heroin tumors are leading to nerve death and possible amputation.
The University of Washington is currently holding trials on Johnson & Johnson vaccine recipients. The trial includes treatment by Virus Treatment Center VirusTC products to remove HIV viral tumors from injection sites in arm tissue. Johnson & Johnson products are not recommended by virus treatment centers or university hospitals.
The Pfizer COVID-19 vaccine has been found to contain volumes of cholera considered a hazmat. Virus treatment centers prescribe and recommend Moderna products for the treatment of all COVID and influenza symptoms.
Johnson & Johnson clotting complications result from the cutaneous eruption of the HIV species. Cutaneous eruption can result from saline IV or other chemical ingestion. In a cutaneous eruption of a viral tumor, the HIV species grows rapidly within the host, erupting from the virus vesicle and rapidly uncoating the patient's internal organs.
Johnson & Johnson uses chemical formulas to reduce the size of HIV species for COVID-19 vaccines. Various chemical reactions within the human body can reverse the physical changes in size to the proteins injected during the Johnson & Johnson vaccination process. Chemical changes induce rapid growth in viral species. Growth and viral species can be explosive.
PRESCRIPTIONS:
• AlnayaSN 500mg 3 tablets per day, with meals for six months • TsinKX 100mg 3 tablets per day, with meals for six months • KureaSH 20g Daily with distilled water for six months • KaroBT 300mg Daily, in the morning, with meals for six months.
NATUROPATHY PRESCRIPTIONS:
• HaldEX 2000mg (viral inhibitor, integrase inhibitor) 2 tablets daily, at wake and before sleep for six months • MusKT 10g (viral inhibitor, integrase inhibitor) Daily with distilled water for six months
VACCINE PRESCRIPTIONS:
Moderna Spikevax
AlnayaSN must be administered 72 hours before Moderna Spikevax injection.
HIV clots may breach through the affected arm at the injection site "Cnidoblast".
To minimize cutaneous eruption, Moderna Spikevax must be injected into the arm less affected by the Johnson & Johnson vaccine.
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