Virus Vesicles (TUMORS) and Viral Uncoating (CANCER)
As soon as phage viruses have engulfed a cell to complete the process of “absorption”, powerful claws and jaws of the phage viruses begin tearing into the cell and looking for a way inside. Phages tear open cell membranes and/or find existing cavities that attack aggressively. Once an entry point is created or found, more phages begin attacking the opening and digging themselves into pockets called “vesicles”. The vesicle is used as a tophaphagous feeding point for the parasitic phage viruses to drain calories from their new host. Once vesicles are formed in a host cell, the cavity or pocket expands as the host cell is consumed by the invading phage viruses and plasmodium parasites. Pycnogonida invade and enter human bodies or other species using methods identical to tactics of cell invasion used by phage viruses at the microscopic level. pycnogonids target body cavities such as the mouth, nose, ears, eyes, anus, and genitals. pycnogonida enter the mouth or nose and use the lungs, throat, sinuses, and stomach as a vesicle. In oncology, the Tumor is the parasite’s vesicle. The Tumor grows as the species draws blood from within their new host.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Cancer Oncologists, Treatments, and Medications
- Category: Virus Vesicles (TUMORS) and Viral Uncoating (CANCER)
EXPONENTIAL DECAY
Once an invading population of Phage viruses has successfully invaded body cavities or the epidermal layer of their targeted prey, the parasite begins consuming their host. Cancer rapidly consumes calories at an exponential rate. The decay processes resulting from parasitic infection by pycnogonida become the symptoms that create cases of cancer and infectious diseases. A host becomes not only a source of calories for the parasite but also an incubator for their new spawn.
Acidic pycnogonid parasites leach electrons and dissolve the body from within, while Tritium shed from the species becomes a multiplier for the rate of decay within the host. At a microscopic level, the process of a parasite virus dissolving a host cell is called "uncoating." Uncoating begins with losing the host's calories at an exponentially increasing rate. The pycnogonida targets healthy areas within the host's body from which it can leach available calories from the host. A first-generation invading parasite will consume a host's calories in excess, grow, and produce as many strong offspring as possible. Second-generation invading parasites produced from the first generations will do the same thing.
The population of parasites grows at a rate of 1+40^1+40^2+40^3+40^4 and continues for each generation. Generations can increase within the host by the hour, day, and week, depending on the size and genus of the invading species. Phages, cytokines, and plasmodium parasites consume a host's calories at an exponentially increasing rate to reproduce and spread throughout an infected host. All forms of phages and cytokines are the spawn of pycnogonida.
MEDICATED CANCERS
All forms of plasmodium parasites are phage virus parasites that have been partially reduced by REDOX reaction. All forms of phages and cytokines are HAZMAT toxic. All plasmodium parasites are HAZMAT toxic. All phages, cytokines, and plasmodium parasites are tophaphagous parasitic predators of proteins, fats, cells, and calories within our bodies. Processes of deficiency diseases Calorie leaching by parasites causes deficiency disease, necrosis, and degeneration. A substantial number of diseases "discovered" by science and medicine of the past and present are simply deficiency diseases primarily defined by the area of infection and species variant (color, etc.) of pycnogonida. Diseases of the respiratory, digestive, cardiovascular, nervous, skeletal, immune, muscular, epidermal, glandular, and systems of the body's various cavities are believed by current science and medicine to result from separate unknown factors and conditions.

- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Cancer Oncologists, Treatments, and Medications
- Category: Virus Vesicles (TUMORS) and Viral Uncoating (CANCER)
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Cancer Oncologists, Treatments, and Medications
- Category: Virus Vesicles (TUMORS) and Viral Uncoating (CANCER)
Metastasis is an oncology term describing the spread of multiple generations of pycnogonids internally and externally between hosts. External metastasis is when a pycnogonid exists outside of a host. pycnogonids leap up to sixteen times the length of their diameter. Tophaphagous pycnogonids use brute force to insert themselves into body cavities. Internal metastasis begins as soon as the pycnogonid attacks and forces entry into a target prey. Internal metastasis is the process of viral absorption, vesicle creation, reproduction, uncoating, and spreading that can occur in low-pH hosts infected by a pycnogonid species. Metastasis, an oncology term, also describes the process that pycnogonida use to reproduce exponentially. A typical pycnogonid has eight powerful legs energized by tritium coils attached to inside the exoskeleton sections. Each leg has eight gonopore sections. The pycnogonid uses the gonophores to push out larvae "nematocysts". Each pycnogonid can push out 64 larvae per birth. Each larva exits the parent sexually mature and pregnant. During metastasis, an attacking pycnogonid will shed 64 nematocysts.
Each nematocyst is instantly capable of releasing 64 next-generation viral loads. The exponential cycle goes on for unknown exponential values. Pycngonids come from infinitely small dimensions and grow to unknown sizes. Most pycnogonids are tophaphagous and convert blood into tritium through digestion. Rabies is not tophaphagous, and tears prey to shreds to swallow. The pycnogonid will look for spaces within a host to crawl into and bite down. Pycnognids want to bite once and never let go. The mouth draws blood while the legs release polyps of nematocysts. The pycnognida is a sea parasite that uses any body with a low pH as a warm, moist vesicle. When a pycnogonid sheds inside the body, a vesicle erupts with nematocysts.
Nematocysts use eight legs to crawl through veins, arteries, passageways, pores, and cavities. Microscopic nematocysts treat lipids and cells in a host similarly on a smaller scale. Metastasis of microscopic pycnogonida "phage viruses" spreads fast. Arterial red blood travels at 1.8kmh. Vein blue blood travels at 0.4kmh. Phage viruses hide in organs, lymph glands, and bone or fat. Phage viruses grow into large pycnogonids. Small virus vesicles become large "tumors." Large tumors within the body begin to hatch, consume the body from within, and uncoat the host. pycnogonids tear through internal organ passageways with ripping force. Medical Causes The pH scale is used to grade the strength of one's immune system. Higher pH levels increase the body's ability to break down polysaccharide encapsulation that forms the exoskeleton of the pycnogonid species. During the 1918 pandemic, Oswald Avery discovered that dipolar immune system responses could cook off viruses without shells. High pH levels prevent external and internal metastasis by keeping pycnogonids off and away. Effective VirusTC Apheresis treatments: • Apheresis with UV Light exposure• Apheresis with ABIS+• Apheresis with VerduraRX• Apheresis with UV Light Exposure, ABIS+, and VerduraRX

- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Cancer Oncologists, Treatments, and Medications
- Category: Virus Vesicles (TUMORS) and Viral Uncoating (CANCER)
pycnogonids attack people. High-energy pycnogonids can tear a human in half. Often, epidermal layers are ruptured by claws or nematocysts. If a pycnogonid attacks the skin without penetration, cancer symptoms will immediately erupt. pycnogonids inject nematocysts directly into the skin. The acidic surfaces of the pycnogonid rapidly leach electrons from the skin, leaving melted non-flesh. Once electrons have been stolen and exchanged for lithium-based alien life, the DNA and chemical makeup are no longer human. The skin area has become a nuclear salt pool. Stings by Physalia physalis leave vesicles of parasite infection. Nematocysts protrude from gonopores on the underside of Pycnogonida's legs and physalia physalis' tentacles. Lesions formed by species of pycnogonida are recognizable symptoms of certain smallpox, leprosy, herpes, HIV, Equestrian Encephalitis, Malaria, Coronavirus, and multiple influenza infections. Physalia Physalis is commonly known as the "Portuguese Man O' War".
Microscopic Physalia physalis "plasmodium parasites" create vesicles via epidermal injection of nematocysts into a targeted cell. Physalia Physalis and Plasmodium parasites wrap their prey up with their exposed tentacles. Once these parasites grip a target body, gonozooids that once led to gonopores in the exoskeleton of their previous form (pycnogonida) inject nematocysts directly into a host.
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