Pycnogonida (PHAGE VIRUS)
The pycnogonida parasite is a lithium-based aquatic crustacean. The phage virus is the youngest and smallest generation of a Pycnogonida’s offspring. The phage virus parasite is tophaphagous, aggressive, intelligent, and parasitic, highly invasive. pycnogonids survive by hunting warm-blooded creatures. A pycnogonid will consume prey that is smaller than itself using powerful jaws to break flesh and drain internal fluids. pycnogonids enter prey larger than itself via available body cavities such as the mouth, nostril, genitalia, anus, eyes, and ears, or penetration of the epidermal layer. Once inside a targeted prey, a pycnogonid attaches itself to internal organs and survives as a parasite. Once pycnogonids parasites enter a targeted host, the species reproduces rapidly at an exponential rate through gonopores on each of the species' eight legs. Phage viruses that spawn from the pycnogonid parasites reproduce at a rate that most host species cannot fight off via a natural immune system response.

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- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
LOW-RESOLUTION VIRUS STEREOCHEMISTRY
The currently accepted theory of what a virus is has been formed and accepted over past generations throughout an ever-evolving field of microscopy. Science currently accepts that a virus cell is a spherical membrane covered by numerous protuberances that come in two shapes. One shape is referred to as a spike, and the other shape is something like a small bundle. Current virus theory accepts that these spherical cells and protuberances found under microscopes are all part of a single species.
HIGH-RESOLUTION STEREOCHEMISTRY
Nearly a century after the Great Influenza outbreak of 1918, the United States Military has industrialized using large-breed phage viruses or pycnogonida. Engineers and scientists have died witnessing the reproductive or spawning habits of the species from a much larger perspective than any microscope could provide during the 1918 pandemic. Tritium is harvested from pycnogonida, which are 20 meters in diameter, on many nuclear aircraft carriers, submarines, and power plants.
Researching phage virus stereochemistry from larger pycnogonida species provides dimensional analysis, high-resolution microscopy, and new insights into what these shapes represent. When viewed as one complete image, the spherical membrane covered in protuberances is a spherical cell that has fallen host to parasitic infection. Instead of a single cell with protuberances, we look at a cell absorbed by octoped phage viruses and plasmodium parasites.

- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
Infections in men and women have been published inaccurately as only capable of transmission via the transfer of bodily fluids. Viruses that create vesicles within reproductive organs are not limited to transfer via bodily fluid, blood-to-blood contact, or sexual transmission. Although microscopic Phages and Plasmodium parasites are often in high concentration in sperm and other bodily fluids, larger pycnogonida may become excited and exit the body via reproductive organs if the host becomes sexually aroused and/or during close intimate contact such as foreplay, etc. Most species of pycnogonida can leap up to 15 times the length of their leg span. All species of pycnogonida are capable of sprint-like speed during crawling movements.
In the 1980s, during the height of the AIDS epidemic, NBA players wisely refused to play basketball with HIV host Magic Johnson to prevent an infection. Dangerous diseases such as HIV and AIDS transfer from person to person via simply crawling out of an infected person's body cavity, traveling down a pant leg, dropping out of a skirt, finding a healthy person to target, and breaching a body cavity such as the mouth, nose, vagina, penis, or anus. A virus can exit a stranger in a restaurant, travel between tables, crawl up your pant leg or dress, and enter you without shaking hands or meeting the person.
Viruses are highly mobile and aggressive. Scientific investigations for pycnogonid toxicity are suppressed to prevent fear of the nuclear industry or "widespread panic." Doctors publish inaccurate claims that close contacts, such as hugs and kisses, cannot transmit HIV. AIDS patients are secretly killed in hospitals when their shedding of the virus becomes too severe. Any warm body with a Ph level below 8.0 is prey for pycnogonids. pycnogonids are drawn to any low-Ph potential host.
All phages, cytokines, and plasmodium parasites will take every opportunity to shed from a dying host and infect a new healthy host as a source of fresh calories needed for further species reproduction. Once a virus population senses the death of a host, the population will exit the dying host in search of a healthy body. Viruses need a living body to survive and are aggressive in attaining one.
HIV can survive outside the body for as long as it can find calories and avoid being killed. If a pycnogonida has been shed from a previous passenger, you can catch a virus infection from someone sitting on a bus seat before you. Transmission only requires that a pycnogonida run up your pant leg or get near your face. pycnogonids target prey and violently leap into any available body cavity.
pycnogonida parasites find suitable prey or new hosts by feeling for "charge” or "Ph level." Overabundances and absences of electrons in nearby lifeforms are critical to the parasites when hunting. Negatively charged "Alkaline” bodies and cells with plenty of electrons present a dangerous reducing agent to pycnogonida parasites and phage viruses. Alkaline bodies are considered a type of chemical predator to the parasite. Positively charged "acidic” bodies and cells lacking electrons offer no danger of chemical reduction to phage virus parasites and become infection hosts or prey.

- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
The current theory on virus "absorption" onto healthy cells involves the use of "spike-shaped" protuberances on the viruses called "hemagglutinin" to bind and latch onto sialic acid "receptors" on the targeted cell-like grappling hooks. As the theory continues, as more hemagglutinin binds to more sialic receptors on the cell, the virus adheres to the body of the targeted cell.
The reality of absorption is that, like any other predator in the wild, the pycnogonida must grip onto areas on its prey where its claws will fit. Once a pycnogonida gets a hold of a host with several of its eight legs, the predator will quickly grab the host with the rest of its appendages and make aggressive maneuvers to breach its prey.
Microscopy is never perfect, as scientists are forced to publish explanations for objects, lifeforms, actions, activities, and processes that they cannot see in clear focus. Fancy terminology like hemagglutinin, sialic acid receptors, and binding of the two only confuse the fact that extremely aggressive eight-legged spider-like creatures are jumping onto cells and wrapping their legs around their target to grab hold wherever their claws will fit.
The tactics of the predatorial approach by the phage species are the same on both living bodies and living cells. During microscopic infancy, phage virus parasites 1/10,000th of a millimeter in diameter do the same thing to cells that a 3dm 3-diameter pycnogonida will do to a fully grown person.
pycnogonida in the wild or running loose in human environments are witnessed using eight legs to crawl, run, leap, swim, and climb toward capturing a host suitable for parasitic infection and reproduction. Suitable hosts at any scale include prey with an acidic or low-Ph level, which will not break down and cook off the pycnogonida via an acid-base REDOX reaction. Hive-minded pycnogonida are highly intelligent and will use all available senses to look for an available host that will not reduce the virus.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
pycnogonids are a highly intelligent, hive-minded species that uses electrical signals generated from Tritium beta decay as a method of communication. Coiled nervous systems found within the species use double latch gates to keep quantum bonds between generations of the species. Physical breakage between an infant pycnogonid’s nervous system and the gonopore of its parent leaves behind chemical bonds. Chemical bonds can transmit electrical signals throughout offspring that are exponentially produced by generations of the species. Electrical signals, data, information, communication, and knowledge received by a single pycnogonida are available to a neural network that expands throughout every connected pycnogonid generation. The neural network available to pycnogonids is larger than any other species found on Earth. pycnogonids were used to transfer qubits through quantum computer networks before Dr. Correo Hofstad developed the quantum snap-circuit at the Pacific National Laboratory.
Exponentially reproducing pycnogonid parasites gain access to an infected host’s brain and neural network. pycnogonids learn (or already know of) the language of signal coding used to command human executive functions. In “Acquired” diseases, a host or host has been demyelinated, and the viral infection has acquired the ability to intercept, interpret, and send electrical signals that control the body. A host of an acquired disease becomes a puppet of varying degrees to a type of “middle-man attack" by a virus population. Schizophrenia, delirium, mental inertia, physical prostration, psychosis, mental collapse, mental disturbances, and general insanity are medical diagnoses that appear after influenza and virus outbreaks.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
The fatty lipid layers that cover and insulate portions of the brain are called Myelin. Myelin, to the function of the nervous system, is like the silicone insulation that surrounds and insulates electrical copper wires and cables and circuit boards to separate individual electric components. Myelin protects and isolates electrical signals produced within the brain. Mylenin protects the brain, stem, spinal cord, and nervous system. Myelin is a source of calories for pycnogonids. Virus infections in the brain target and consume the fatty lipid content that insulates neurons, dendrites, and nerve bundles in the white matter of the cerebrum.
When Myelin, which protects the brain, is consumed in a process called Demyelination, electrical signals within the nervous system are unprotected. Degeneration of signal power and quality of transmissions throughout the nervous system are standard systems of demyelination. Demylenation is similar to symptoms of decay found on insulation within electrical systems and devices. Acute demyelination of the nervous system can lead to more severe short circuits within the nervous system of a host of a virus infection. Acute demyelination or short-circuiting of the nervous system is often responsible for seizures, tremors, shaking, spasms, uncontrollable muscle movement, physical impairment, mental degradation, and trouble during speech. Multiple Sclerosis M.S., Parkinson’s, and epilepsy are symptoms of demyelination by a pycnogonid parasite.
demyelination can only be halted and reversed by removing the virus infection from the body and consuming massive volumes of regenerative supplements such as collagen, MSM, glucosamine, and amino acids. Zinc is used by the body to build and repair neurons and nerves within your body. These regenerative supplements help rebuild the Myelin lipid content throughout the nervous system. High-Ph alkaline treatments and a high intake of regenerative supplement-rich diets can repair and stimulate Myelin growth needed for rebuilding and supporting a healthy nervous system.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
phage virus parasites attack, destroy, and manipulate the brain, spine, and nervous system in hosts that they infect. During autopsies of hosts of lethal viruses, the proteins, cytokines, macrophages, granulocytes, plasmodium parasites, and their various Genera are found within the white matter of the cerebrum, in the meninges that surround the brain, and within the spinal cord.
The fatty lipid layers that cover and insulate portions of the brain are called Myelin. Myelin, to the function of the nervous system, is like the silicone insulation that surrounds and insulates electrical copper wires and cables or is used in circuit boards to separate individual electric components. Myelin protects and isolates electrical signals produced within the brain, which travel down the spinal cord and throughout the nervous system. As for viruses, Mylin has only one function: a source of available calories as a food supply.
In the brain, virus infections target and consume the fatty lipid content that surrounds and insulates neurons, dendrites, and nerve bundles contained within the white matter of the cerebrum. When Myelin, which protects the brain, is consumed in a process called demyelination, electrical signals within the nervous system are unprotected. Degeneration of signal power and quality of transmissions throughout the nervous system are standard systems of demyelination that resemble symptoms of decay found on insulation within electrical systems and devices resulting from rodent infestations.
Acute demyelination of the nervous system can lead to more severe short circuits within the nervous system of a host of a virus infection. Acute demyelination or short-circuiting of the nervous system is often responsible for seizures, tremors, shaking, spasms, uncontrollable muscle movement, physical impairment, mental degradation, and trouble during speech found as epidemic diseases such as M.S., Parkinson's, and epilepsy. demyelination can only be halted and reversed via removing the virus infection from the body and consuming massive volumes of regenerative supplements such as collagen, MSM, glucosamine, and amino acids. Zinc is used by the body to build and repair neurons and nerves within your body. These regenerative supplements help rebuild the Myelin lipid content throughout the nervous system. High-Ph alkaline treatments and a high intake of regenerative supplement-rich diets can repair and stimulate Myelin growth needed for rebuilding and supporting a healthy nervous system.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
Infections by pycnogonids are often responsible for most reproductive cancers including ovarian cancer, and prostate cancer. pycnogonid infections of reproductive organs are responsible for sexually transmitted diseases such as HIV, AIDS, HPV, Herpes, Gonorrhea, Crab, Syphilis, etc. Symptoms of pycnogonid infection of the genitalia in the form of reproductive organ infection and/or cancers include pycnogonida exiting or shedding from the penis in men or vagina in females. Additionally, pycnogonida may become excited and exit the body via reproductive organs if the host becomes sexually aroused and/or during close intimate contact such as foreplay, etc. Sores, scabs, and scars found on the exterior epidermal layers of male and female genitalia often result from pycnogonid bites and/or areas in which a nematocyst has penetrated the epidermis to create a vesicle area. Phages, Plasmodium parasites are often in high concentration within sperm and other bodily fluids associated with sexual intercourse and reproduction. In males and females, “outbreak” or “flare-up” scenarios can occur when a viral load reproduces heavily and exits the body cavities en mass. In similar scenarios, a sudden rise in an infected host’s Ph level will cause an exodus of pycnogonida from reproductive organs which from an outside perspective resemble an outbreak or flare-up.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
Viruses are attracted to bone marrow and will reproduce rapidly once the skeleton is breached, leading to several forms of leukemia. Disease, organ failure, and cancer are a result of infections within the body by pycnogonids, Physalia physalis, Phage Viruses, and Plasmodium Parasites. Tetanus, Meningitis, Necrosis, and degeneration of the muscular systems can result from uncoating a host’s muscular proteins.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
Phages, Cytokines, macrophages, Granulocytes, and RNA at their smallest detectible generation hunt and consume calories from the Circulatory system, bloodstream, digestive system, bone marrow, lymphoid tissue, liver, spleen, the white matter of the cerebrum, and the meninges surrounding the brain. The Process of Respiratory Coronaviruses and COVID Infections pycnogonida infections of respiratory systems such as the throat and lungs are often diagnosed as diseases such as Coronavirus, COVID, SARS, Tuberculosis, Influenza such as H1N1, Cholera, emphysema, pneumonia, cyanosis, ARDS, and Bubonic Plague. These diseases result from pycnogonids, which have absorbed a targeted host, breached the oral or nasal cavities, creating a vesicle in the lungs, and dissolving or uncoating tissue within the lungs. pycnogonids often attach themselves within the upper respiratory tract, below the larynx. Offspring of the first-generation invader, including microscopic phage viruses, attack epithelial cells within the upper respiratory tract and use them as calories and incubators for producing microscopic virus proteins. Often, within ten hours of an attack by a phage virus, an epithelial cell will uncoat and burst open to release between 1,000 and 10,000 new virus cells. Epithelial cells make up the insulation or lining of the entire respiratory tract.
The throat becomes stripped and raw as a virus infection consumes a host's epithelial cells. A sore throat is often the first sign of a viral infection. Hosts usually begin feeling symptoms during the fifth or sixth generation of the virus' reproduction. Suppose the host's immune system cannot break down and kill a virus infection while incubating within the upper respiratory system. In that case, proteins will continue to breed and spread downward into the lungs. "Acute inflammatory injection (AII)" is the process of rapid necrosis of the epithelial lining of the bronchial tree. pycnogonids and their microscopic phage offspring rip apart capillaries and bronchioles within the lungs. Lungs become clogged with growing virus proteins, blood, fluids, and scar tissues. Essential "surfactant" within the lungs disappears, and the function of oxygen absorption rapidly decreases. Extreme distress in the lungs, termed "acute respiratory distress syndrome (ARDS)," is a process with no cure. Today's intensive care units administer oxygen to ARDS patients to keep hosts alive until they can recover. Coronaviruses such as the 2003 outbreak of "severe acute respiratory syndrome (SARS)" and the 2019 outbreak of COVID-19 kill humans via ARDS. In ARDS, hosts' organs fail due to lack of oxygen, and fluids fill the lungs and leach into the heart, causing strain on the heart or causing breathing stops. Viral infection of the respiratory tract may also lead to bacterial pneumonia. A lack of epithelial cells destroys a host's ability to clear the respiratory tract of bacteria.
pycnogonids, virus proteins, and lethal bacteria flow freely into the lungs. ARDS becomes pneumonia when the lungs "consolidate" or become hard, solid, stiff, and inelastic. Pneumonia is often a result of an infection of a bacteria called pneumococci. Pneumonia frequently kills by restricting the flow of oxygen into the host or by creating fissures, cracks, and sores in stiff lung tissue, which allows virus proteins and bacteria to enter the host's bloodstream. End-of-Life Degeneration Viruses invade internal organs, destroy tissue, and consume the body's proteins. Reye's syndrome and other diseases lead to liver failure. Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands, leading to breakdowns in enzyme and hormone production and regulatory functions.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
Once a virus infection enters the bloodstream, phage viruses and their plasmodium parasites absorb the arteries. pycnogonids will spread, form vesicles, and uncoat a host’s internal organs. Infection of the bloodstream can lead to hemorrhagic diseases like epistaxis. Epistaxis results from the rapid uncoating of veins, arteries, and blood vessels by phage viruses that grow too large within the cardiovascular system to support life within the infected host. When the cardiovascular system uncoats, the mouth, nose, eyes, skin, Gentelia, anus, and other body cavities begin hemorrhaging. Hyperemia leaves the brain flat and dry when blood begins flooding the brain. Blood infections such as HIV, AIDS, Hepatitis, EEE, etc., are often transferred between humans via narcotics, blood transfusions, transplants, food contamination, blood-to-blood contact, ectoparasites, etc.
- Details
- Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv
- Parent Category: Virus Pathologists, Treatments, and Medications
- Category: Pycnogonida (PHAGE VIRUS)
These parasites cause sharp pain with their eight claws and jaws as they feed on the walls of a host's stomach and small intestine. Microscopic phages of the pycnogonida can enter the bloodstream and various organs through the digestive system. Signs of a digestive system infection may include pycnogonida, which travel up the esophagus, through the throat, and out of the mouth, or those that appear in the stool during bowel movements. pycnogonida infections in the digestive system cause most stomach and bowel cancers. Processes of consumption, disease, and cancer of the internal organs Viruses attack the cardiovascular system, invade internal organs, destroy tissue, and consume the body's proteins. Reye's syndrome and other diseases lead to liver failure.
Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands, leading to breakdowns in enzyme and hormone production and regulatory functions. Viruses are attracted to bone marrow and will reproduce rapidly once the skeleton is breached, leading to several forms of leukemia. Disease, organ failure, and cancers result from infections within the body by pycnogonids, Physalia Physalis, Phage Viruses, and Plasmodium Parasites. Radiation sickness and tissue destruction: All phages, cytokines, and plasmodium parasites emit beta-radioactive Tritium into the body cavities, vesicles, bloodstream, and respiratory system of an infected host. Beta-decay of Tritium from phages, cytokines, and plasmodium parasites causes radiation sickness in an infected host, often associated as a symptom of a particular disease.
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