The previously accepted theory of what a virus is had been formed and accepted via past generations throughout an ever-evolving field of Microscopy. Science currently accepted that a virus cell is a spherical membrane covered by numerous protuberances that come in two shapes. One shape is referred to as a spike and the other shape is referred to as something like a small bundle. Previous virus theory accepts that these spherical cells and protuberances found under microscopes are all part of a single species.

Nearly a century after the great influenza outbreak of 1918, the United States Military has industrialized the use of large-breed phage viruses or pycnogonida. Engineers and scientists have died witnessing the reproductive or spawning habits of the species from a much larger perspective than any microscope could provide during the 1918 pandemic. Tritium is harvested from pycnogonida which are 20 meters in diameter on many nuclear aircraft carriers, submarines, and power plants. Researching phage virus stereochemistry from larger pycnogonida species provides dimensional analysis, high-resolution microscopy, and new insights into what these shapes represent. When viewed as one complete image, the spherical membrane covered in protuberances is a spherical cell that has fallen host to parasitic infection. Instead of a single cell with protuberances, we are looking at a cell being absorbed by octoped phage viruses and plasmodium parasites.

The phage virus parasite consists of a crustaceous body with an exoskeleton composed of a sugary carbohydrate. In 1917 Oswald Avery of the Rockefeller Institute first discovered that pneumococci phages were surrounded by a capsule of polysaccharides, a pure carbohydrate sugar shell. In Avery’s first research paper on the subject, he investigated these “specific soluble substances”. The crustaceous shell of the phage virus parasite is the polysaccharide capsule of the phage virus. In both the large breed pycnogonida and the microscopic phage virus, the composition and stereochemistry of the lifeform is the same. The exoskeleton of the phage virus parasite is a low-Ph acetic sugar that provides physical protection to the internal gelatinous mass of the species. The exoskeleton becomes tempered at elevated temperatures and is extraordinarily strong. In a parasitic infection of any phage virus parasite, the exoskeleton is nearly impenetrable to the immune system of a host. Although the exoskeleton of the phage virus parasite provides great physical tensile strength, the chemical composition of low Ph acetic sugar carbohydrates is the species' primary weakness. In an alkaline environment, or when in contact with an alkaline substance, the polysaccharide capsule or exoskeleton of the phage virus parasite completely dissolves. Upon dissolution of the carbohydrate exoskeleton/polysaccharide capsule of the phage virus parasite species, a gelatinous globule of purple-colored mass of internal organs will remain. This purple gelatinous mass is the physalia physalis, known in pathology terms as the plasmodium parasite. During the great influenza of 1918, it was discovered that the immune system could not attack pneumococci surrounded by capsules, but easily destroyed pneumococci without capsules. Exactly one century before the worldwide epidemic of COVID-19, pneumococci with intact exoskeletons were growing rapidly in the lungs of their hosts and killing within weeks, days, and even hours.

When an alkaline environment or substance reduces the oxidized polysaccharide hydrocarbonate capsulized exoskeleton of the phage virus parasite, the internal mass that remains is the plasmodium parasite. Sections of the phage virus parasite's legs, body, head, tail, and ovigars dissolve via REDOX reactions quickly within minutes when in contact with a reducing agent. Complex coil groups once anchored to points of the exoskeleton become one big hanging mess. Dissolving the oxidized shell, exoskeleton, or polysaccharide capsule of the large breed pycnogonida or microscopic phage virus, leaves the large breed physalia physalis or microscopic plasmodium parasite. The damage done to the coils and anchor points within the exoskeleton and the bloating of the bladder of the resulting physalia physalis during a REDOX reaction is violent. In a liquid or aquatic environment, the coil structures that are externally visible in the physalia physalis species immediately become intertwined in a randomly contorted mess. The randomly intertwined bundle of coils hangs from a balloon-like body which expands after losing the pressurized capsule or exoskeleton.

Scientists Oswald Avery and Fred Griffith published theories that pneumococci species with dissolved capsules could somehow be observed under the microscope transforming into species with intact capsules. Modern observations of physalia physalis without an oxidized exoskeleton or hydrocarbonate polysaccharide capsule have revealed the birth of new spawns of phage virus parasite from physalia physalis. physalia physalis give birth to new phage virus parasites with intact capsules in the form of nematocysts which spawn from select coils protruding from the blue-purple gelatinous mass. Infant phages (Nematocysts) hatch from gonopores within the exposed coils. Coils that come in contact with a species of prey can inject nematocysts into a host via a “sting” which allows the infant nematocysts to grow as a parasite infection, further spreading the species and continuing its lifespan. A phage virus parasite that loses its protective structure from contact with a reducing agent. may release millions to billions of smaller physalia physalis and plasmodium parasites which all simultaneously lose their protective structures. Each generation of damaged physalia physalis has within its reproductive coils, new generations of intact phage virus parasites. Larval phage virus parasites within physalia physalis reproductive coils are called Nematocysts. Each generation of phage virus parasite produces new spawns from its gonopores simultaneously.

In an intact phage virus parasite, reproductive coils lead to gonopores in the legs of the species. Gonopores are exposed openings that give birth to exponentially polyping generations of new phage virus parasites. A single gonopore exists on each section of every leg, between each joint. With eight legs, each having five sections, the pycnogonida has 40 gonopores. Every gonopore can simultaneously spawn a single phage virus parasite. Every generation of newly spawned phage virus parasite grows from its parent's gonopores until it is released, often occurring only in the presence of survivable habitat or targeted host for parasite invasion. Each generation of spawned phage virus parasite is capable of spawning newer generations from its gonopores, even while still attached and growing from its own parent’s leg. In effect, a first-generation pycnogonida with a 3-meter leg span may spawn 40 attached pycnogonida offspring with leg spans of 2 decimeters. 40 second-generation 2dm pycnogonida may simultaneously spawn 1600 attached pycnogonida with a 2cm leg span. The 1600 third-generation pycnogonida may spawn 64,000 attached pycnogonida with a 2mm leg span. The 64,000 fourth-generation pycnogonida may spawn 2,560,000 attached phage virus parasites with a 200-micrometer leg span. The 2,560,000 fifth-generation phage virus parasites may spawn 102,400,000 attached phage virus parasites with a leg span of 20 micrometers. The 102,400,000 sixth- generation phage virus parasites may spawn 4,096,000,000 microphages. ECNM HOFSTAD This biological method of exponential reproduction is how viruses spread so far and so fast in short periods. A large breed pycnogonida enters a host prey and immediately releases all generations of spawn into the host’s body to spread, grow, and reproduce. This method of parasitic infection is highly effective at surviving on a host’s blood, and organs until the species can be spread throughout the host species’ population.