VIRAL UNCOATING (CANCER)

Once an invading population of Phage viruses has successfully invaded body cavities or the epidermal layer of their targeted prey, the parasite begins consuming their host. Cancer rapidly consumes calories at an exponential rate. The processes of decay that result from parasitic infection by pycnogonida become the symptoms that create cases of cancer and infectious diseases. A host becomes not only a source of calories for the parasite but also an incubator for their new spawn. Acidic pycnogonid parasites leach electrons and dissolve the body from within, while Tritium shed from the species becomes a multiplier for the rate of decay within the host. At a microscopic level, the process of a parasite virus dissolving a host cell is called “uncoating”. Uncoating begins with a loss of the host’s calories at an exponentially increasing rate. The pycnogonida targets healthy areas within the host’s body from which it can leach available calories from the host. A first-generation invading parasite will consume a host’s calories in excess, grow, and produce as many strong offspring as possible. Second-generation invading parasites produced from the first generations will do the same thing. The population of parasites grows at a rate of 1+40^1+40^2+40^3+40^4 and continues for each generation. Generations can increase within the host by the hour, day, and week depending on the size and genus of the invading species. Phages, cytokines, and plasmodium parasites consume a host’s calories at an exponentially increasing rate to exponentially reproduce and spread throughout an infected host. All forms of phages and cytokines are the spawn of pycnogonida.

All forms of plasmodium parasites are forms of phage virus parasites that have been partially reduced by REDOX reaction. All forms of phages and cytokines are HAZMAT toxic. All plasmodium parasites are HAZMAT toxic. All forms of phages, cytokines, and plasmodium parasites are tophaphagous parasitic predators of proteins, fats, cells, and calories within our bodies. Processes of deficiency diseases Calorie leaching by parasites causes conditions of deficiency disease, necrosis, and degeneration. A substantial number of diseases “discovered” by science and medicine of the past and present are simply deficiency diseases defined largely by the area of infection and species variant (color, etc.) of pycnogonida. Diseases of the respiratory system, digestive system, cardiovascular system, nervous system, skeletal system, immune system, muscular system, epidermal system, glandular systems, and systems of the body’s various cavities are believed by current science and medicine to result from separate unknown factors and conditions.

Nearly all deficiency diseases of human systems are symptoms of parasite infection by species of pycnogonida. Processes of diseases of the bloodstream Once a virus infection enters the bloodstream, phage viruses, and their plasmodium parasites begin absorbing, forming vesicles, and uncoating a host’s internal organs. Infection of the bloodstream can lead to diseases such as hemorrhagic diseases like epistaxis. Epistaxis results from the rapid uncoating of veins, arteries, and blood vessels by phage viruses which grow too large within the cardiovascular system to support life within the infected host. When the cardiovascular system uncoats, internal hemorrhaging and external hemorrhaging from the mouth, nose, eyes, skin, Gentelia, anus, and other body cavities result. Blood flooding the brain, termed hyperemia leaves the brain flat and dry. Blood infections of pycnogonida or Phage viruses such as HIV, AIDS, Hepatitis, EEE, etc. Are often transferred between humans via blood-to-blood contact such as mosquito bites, needle sharing, blood transfusions, etc. Processes of disease within the digestive system pycnogonida which contaminate food, and water supplies enter a host’s stomach and spread throughout the digestive system.

These parasites cause sharp pain with their eight claws and sharp jaws as they feed on the walls of a host’s stomach and small intestine. Microscopic phages of the pycnogonida can enter the bloodstream and various organs through the digestive system. Signs of a digestive system infection may include pycnogonida which travel up the esophagus, through the throat, and out of the mouth, or those that appear in the stool during bowel movements. pycnogonida infections in the digestive system stand for most of the stomach and bowel cancers. Processes of consumption, disease, and cancer of the internal organs Viruses attack the cardiovascular system, invade internal organs, destroy tissue, and consume the body’s proteins. Reye’s syndrome and other diseases lead to liver failure. Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands leading to breakdowns in enzyme and hormone production as well as regulatory functions. Viruses are attracted to bone marrow and will reproduce rapidly once the skeleton is breached, leading to several forms of leukemia. Disease, organ failure, and cancers are the results of infections within the body by pycnogonids, Physalia Physalis, Phage Viruses, and Plasmodium Parasites. Radiation sickness and the destruction of tissue All phages, cytokines, and plasmodium parasites emit beta-radioactive Tritium into the body cavities, vesicles, bloodstream, and respiratory system of an infected host. Beta-decay of tritium from phages, cytokines, and plasmodium parasites causes ECNM HOFSTAD radiation sickness in an infected host which is often associated as a symptom of a particular disease.

Phages, Cytokines, macrophages, Granulocytes, and RNA at their smallest detectible generation hunt and consume calories from the Circulatory system, bloodstream, digestive system, bone marrow, lymphoid tissue, liver, spleen, the white matter of the cerebrum, and the meninges surrounding the brain. The Process of Respiratory Coronaviruses and COVID Infections pycnogonida infections of respiratory systems such as the throat and lungs are often diagnosed as diseases such as Coronavirus, COVID, SARS, Tuberculosis, Influenza such as H1N1, Cholera, emphysema, pneumonia, cyanosis, ARDS, and Bubonic Plague. These diseases are resultant of pycnogonids which have absorbed a targeted host, breached the oral or nasal cavities, created a vesicle in the lungs, and are dissolving or uncoating tissue within the lungs. pycnogonids often attach themselves within the upper respiratory tract, below the larynx. Offspring of the first-generation invader, including microscopic phage viruses, attack epithelial cells within the upper respiratory tract and use them as calories and as incubators for the production of microscopic virus proteins. Often within ten hours of an attack by a phage virus, an epithelial cell will uncoat and burst open to release between 1,000 and 10,000 new virus cells. Epithelial cells make up the insulation or lining of the entire respiratory tract.

As a virus infection consumes a host’s epithelial cells, the throat becomes stripped and raw. A sore throat is often the first sign of a viral infection. Hosts often begin feeling symptoms during the fifth or sixth generation of the virus’ reproduction. If the host’s immune system cannot break down and kill a virus infection during its incubation within the upper respiratory system, proteins will continue to breed and spread downward into the lungs themselves. “Acute inflammatory injection (AII)” is the process of rapid necrosis of the epithelial lining of the bronchial tree. pycnogonids and their microscopic phage offspring begin ripping apart capillaries and bronchioles within the lungs. Lungs become clogged with growing virus proteins, blood, fluids, and scar tissues. Essential “surfactant” within the lungs disappears and the function of oxygen absorption within the lungs rapidly decreases. Extreme distress in the lungs termed “acute respiratory distress syndrome (ARDS)” is a process with no cure. Today’s intensive care units administer oxygen to ARDS patients to keep hosts alive until they can recover. Coronaviruses such as the 2003 outbreak of “severe acute respiratory syndrome (SARS)” and the 2019 outbreak of COVID-19 kill humans via ARDS. In ARDS, hosts' organs fail due to lack of oxygen, fluids in the lungs leach into the heart causing strain on the heart or breathing just stops. Viral infection of the respiratory tract may also lead to bacterial pneumonia. A lack of epithelial cells destroys a host’s ability to clear the respiratory tract of bacteria.

pycnogonids, virus proteins, and lethal bacteria flow freely into the lungs. ARDS becomes pneumonia when the lungs “consolidate” or become hard, solid, stiff, and inelastic. Pneumonia is often a result of an infection of a bacteria called pneumococci. Pneumonia often kills by either restricting the flow of oxygen into the host, or by the creation of fissures, cracks, and sores in stiff lung tissue which allows virus proteins and bacteria into the host’s bloodstream. End-of-Life Degeneration Viruses invade internal organs, destroy tissue, and consume the body’s proteins. Reye’s syndrome and other diseases lead to liver failure. Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands leading to breakdowns in enzyme and hormone production as well as regulatory functions.

Viruses are attracted to bone marrow and will reproduce rapidly once the skeleton is breached, leading to several forms of leukemia. Disease, organ failure, and cancers are a result of infections within the body by pycnogonids, Physalia Physalis, Phage Viruses, and Plasmodium Parasites. Tetanus, Meningitis, Necrosis, and degeneration of the muscular systems can result from the uncoating of a host’s muscular proteins.

Cyanosis develops in the epidermal layer as the death of portions of the body results from a lack of oxygen flow in the blood and the body turns black with the appearance of frostbite. Emphysema and Smallpox boils appear at the surface of the host’s skin as air channels form in the body from open lesions and cracks in the lungs. These air channels allow air to escape the lungs and form pockets under the skin where viruses produce new vesicles. Phage viruses may travel through these oxygen fissures and burst from Smallpox sores when the host is near death. When these pockets burst, the skin rips open, further exposing the body to external infection while simultaneously shedding virus proteins from within. Headaches of the ear, sinus, and eyes result from inflammation of the inner ear and sinus cavities as the body tries to purge oversized virus proteins through mucus channels which become a clogged bottleneck. These sinus and mucus channels bloat with intense pressure and can lead to otitis media and subconjunctival hemorrhaging. STDs are symptoms of body cavities being used as vesicles for pycnogonids. Phage Viruses often enter a host via the genitalia in males and females. While female reproductive cavities may present an easier target for varied sizes of pycnogonida, male genitalia become a direct passage for the transfer of smaller pycnogonids during sexual intercourse. In females, pycnogonids often create vesicles in the uterus, and in males, pycnogonids will often create vesicles in the prostate.

Infections by pycnogonids are often responsible for most reproductive cancers including ovarian cancer, and prostate cancer. pycnogonid infections of reproductive organs are responsible for sexually transmitted diseases such as HIV, AIDS, HPV, Herpes, Gonorrhea, Crab, Syphilis, etc. Symptoms of pycnogonid infection of the genitalia in the form of reproductive organ infection and/or cancers include pycnogonida exiting or shedding from the penis in men or vagina in females. Additionally, pycnogonida may become excited and exit the body via reproductive organs if the host becomes sexually aroused and/or during close intimate contact such as foreplay, etc. Sores, scabs, and scars found on the exterior epidermal layers of male and female genitalia often result from pycnogonid bites and/or areas in which a nematocyst has penetrated the epidermis to create a vesicle area. Phages, Plasmodium parasites are often in high concentration within sperm and other bodily fluids associated with sexual intercourse and reproduction. In males and females, “outbreak” or “flare-up” scenarios can occur when a viral load reproduces heavily and exits the body cavities en mass. In similar scenarios, a sudden rise in an infected host’s Ph level will cause an exodus of pycnogonida from reproductive organs which from an outside perspective resemble an outbreak or flare-up.

phage virus parasites attack, destroy, and manipulate the brain, spine, and nervous system in hosts that they infect. During autopsies of hosts of lethal viruses, the proteins, cytokines, macrophages, granulocytes, plasmodium parasites, and their various Genera are found within the white matter of the cerebrum, in the meninges that surround the brain, and within the spinal cord.

The fatty lipid layers that cover and insulate portions of the brain are called Myelin. Myelin, to the function of the nervous system, is like the silicone insulation that surrounds and insulates electrical copper wires and cables or and used in circuit boards to separate individual electric components. Myelin protects and isolates electrical signals produced within the brain which travel down the spinal cord and throughout the nervous system. As for viruses, Mylin has only one function, being a source of available calories as a food supply. In the brain, virus infections target and consume the fatty lipid content that surrounds and insulates neurons, dendrites, and nerve bundles contained within the white matter of the cerebrum. When Myelin, which protects the brain, is consumed in a process called Demyelination, electrical signals within the nervous system are unprotected.

Degeneration of signal power and quality of transmissions throughout the nervous system are common systems of demyelination that resemble symptoms of decay found on insulation within electrical systems and devices resulting from rodent infestations. Acute demyelination of the nervous system can lead to more severe short circuits within the nervous system of a host of a virus infection. Acute demyelination or short-circuiting of the nervous system is often responsible for seizures, tremors, shaking, spasms, uncontrollable muscle movement, physical impairment, mental degradation, and trouble during speech found as epidemic diseases such as M.S., Parkinson’s, and epilepsy. demyelination can only be halted and reversed via the removal of the virus infection from the body and the consumption of massive volumes of regenerative supplements such as collagen, MSM, glucosamine, and amino acids. Zinc is used by the body to build and repair neurons and nerves within your body. These regenerative supplements help rebuild the Myelin lipid content throughout the nervous system. High-Ph alkaline treatments and a high intake of regenerative supplement-rich diets can repair and stimulate Myelin growth needed for rebuilding and supporting a healthy nervous system.