RESPIRATORY SYSTEM CANCERS
pycnogonids often attach themselves within the upper respiratory tract, below the larynx. Offspring of the first-generation invader, including microscopic phage viruses, attack epithelial cells within the upper respiratory tract and use them as calories and incubators for producing microscopic virus proteins. Often, within ten hours of an attack by a phage virus, an epithelial cell will uncoat and burst open to release between 1,000 and 10,000 new virus cells. Epithelial cells make up the insulation or lining of the entire respiratory tract.
The Process of Respiratory Coronaviruses and COVID Infections Pycnogonida infections of respiratory systems such as the throat and lungs are often diagnosed as diseases such as Coronavirus, COVID, SARS, Tuberculosis, Influenza such as H1N1, Cholera, emphysema, pneumonia, cyanosis, ARDS, and Bubonic Plague. These diseases result from pycnogonids that have absorbed the body, breached the oral or nasal cavities, created a vesicle in the lungs, and dissolved or uncoated tissue within the lungs. The throat becomes stripped and raw as a virus infection consumes a host’s epithelial cells. A sore throat is often the first sign of a viral infection. Hosts often begin feeling symptoms during the fifth or sixth generation of the virus’ reproduction. If the host’s immune system cannot break down and kill a virus infection during its incubation within the upper respiratory system, proteins will continue to breed and spread downward into the lungs. “Acute inflammatory injection (AII)” is the process of rapid necrosis of the epithelial lining of the bronchial tree. pycnogonids and their microscopic phage offspring rip apart capillaries and bronchioles within the lungs. Lungs become clogged with growing virus proteins, blood, fluids, and scar tissue. Essential “surfactant” within the lungs disappears, and the function of oxygen absorption rapidly decreases.
Extreme distress in the lungs, termed “acute respiratory distress syndrome (ARDS)”, is a process that had no cure before 2023’s Operation Cancer Moonshot. Today’s intensive care units administer oxygen to ARDS patients to keep hosts alive until they can recover with VirusTC products. Coronaviruses such as the 2003 outbreak of “severe acute respiratory syndrome (SARS)” and the 2019 outbreak of COVID-19 kill humans via ARDS. In those with ARDS, organs fail due to a lack of oxygen when fluids in the lungs leach into the heart. ARDS bursts through the heart or the lungs. Viral infection of the respiratory tract may also lead to bacterial pneumonia. A lack of epithelial cells destroys a host’s ability to clear bacteria from the respiratory tract.
pycnogonids, virus proteins, and lethal bacteria flow freely into the lungs. ARDS becomes pneumonia when the lungs “consolidate” or become hard, solid, stiff, and inelastic. Pneumonia is often a result of an infection of a bacteria called pneumococci. Pneumonia often kills by restricting the flow of oxygen into the host or by creating fissures, cracks, and sores in stiff lung tissue, which allows virus proteins and bacteria to enter the host’s bloodstream. End-of-Life Degeneration Viruses invade internal organs, destroy tissue, and consume the body’s proteins. Reye’s syndrome and other diseases lead to liver failure. Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands, leading to breakdowns in enzyme and hormone production and regulatory functions.