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Pycnogonida (PHAGE VIRUS)

The pycnogonida parasite is a lithium-based aquatic crustacean. The phage virus is the youngest and smallest generation of a Pycnogonida’s offspring. The phage virus parasite is tophaphagous, aggressive, intelligent, and parasitic, highly invasive. pycnogonids survive by hunting warm-blooded creatures. A pycnogonid will consume prey that is smaller than itself using powerful jaws to break flesh and drain internal fluids. pycnogonids enter prey larger than itself via available body cavities such as the mouth, nostril, genitalia, anus, eyes, and ears, or penetration of the epidermal layer. Once inside a targeted prey, a pycnogonid attaches itself to internal organs and survives as a parasite. Once pycnogonids parasites enter a targeted host, the species reproduces rapidly at an exponential rate through gonopores on each of the species' eight legs. Phage viruses that spawn from the pycnogonid parasites reproduce at a rate that most host species cannot fight off via a natural immune system response.

Details: Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv; Parent Category: Virus Pathologists, Treatments, and Medications; Category: Pycnogonida (PHAGE VIRUS)

phage virus parasites attack, destroy, and manipulate the brain, spine, and nervous system in hosts that they infect. During autopsies of hosts of lethal viruses, the proteins, cytokines, macrophages, granulocytes, plasmodium parasites, and their various Genera are found within the white matter of the cerebrum, in the meninges that surround the brain, and within the spinal cord.

The fatty lipid layers that cover and insulate portions of the brain are called Myelin. Myelin, to the function of the nervous system, is like the silicone insulation that surrounds and insulates electrical copper wires and cables or is used in circuit boards to separate individual electric components. Myelin protects and isolates electrical signals produced within the brain, which travel down the spinal cord and throughout the nervous system. As for viruses, Mylin has only one function: a source of available calories as a food supply.

In the brain, virus infections target and consume the fatty lipid content that surrounds and insulates neurons, dendrites, and nerve bundles contained within the white matter of the cerebrum. When Myelin, which protects the brain, is consumed in a process called Demyelination, electrical signals within the nervous system are unprotected. Degeneration of signal power and quality of transmissions throughout the nervous system are standard systems of demyelination that resemble symptoms of decay found on insulation within electrical systems and devices resulting from rodent infestations.

Acute demyelination of the nervous system can lead to more severe short circuits within the nervous system of a host of a virus infection. Acute demyelination or short-circuiting of the nervous system is often responsible for seizures, tremors, shaking, spasms, uncontrollable muscle movement, physical impairment, mental degradation, and trouble during speech found as epidemic diseases such as M.S., Parkinson's, and epilepsy. demyelination can only be halted and reversed via removing the virus infection from the body and consuming massive volumes of regenerative supplements such as collagen, MSM, glucosamine, and amino acids. Zinc is used by the body to build and repair neurons and nerves within your body. These regenerative supplements help rebuild the Myelin lipid content throughout the nervous system. High-Ph alkaline treatments and a high intake of regenerative supplement-rich diets can repair and stimulate Myelin growth needed for rebuilding and supporting a healthy nervous system.

Read more: BONE & MUSCLE CANCERS

Details: Written by: Dr. Correo “Cory” Andrew Hofstad J.S.D MPHJD MSPH JD Ph.D. M.D. D.O. MBACOGS MDiv; Parent Category: Virus Pathologists, Treatments, and Medications; Category: Pycnogonida (PHAGE VIRUS)

Phages, Cytokines, macrophages, Granulocytes, and RNA at their smallest detectible generation hunt and consume calories from the Circulatory system, bloodstream, digestive system, bone marrow, lymphoid tissue, liver, spleen, the white matter of the cerebrum, and the meninges surrounding the brain. The Process of Respiratory Coronaviruses and COVID Infections pycnogonida infections of respiratory systems such as the throat and lungs are often diagnosed as diseases such as Coronavirus, COVID, SARS, Tuberculosis, Influenza such as H1N1, Cholera, emphysema, pneumonia, cyanosis, ARDS, and Bubonic Plague. These diseases result from pycnogonids, which have absorbed a targeted host, breached the oral or nasal cavities, creating a vesicle in the lungs, and dissolving or uncoating tissue within the lungs. pycnogonids often attach themselves within the upper respiratory tract, below the larynx. Offspring of the first-generation invader, including microscopic phage viruses, attack epithelial cells within the upper respiratory tract and use them as calories and incubators for producing microscopic virus proteins. Often, within ten hours of an attack by a phage virus, an epithelial cell will uncoat and burst open to release between 1,000 and 10,000 new virus cells. Epithelial cells make up the insulation or lining of the entire respiratory tract.

The throat becomes stripped and raw as a virus infection consumes a host's epithelial cells. A sore throat is often the first sign of a viral infection. Hosts usually begin feeling symptoms during the fifth or sixth generation of the virus' reproduction. Suppose the host's immune system cannot break down and kill a virus infection while incubating within the upper respiratory system. In that case, proteins will continue to breed and spread downward into the lungs. "Acute inflammatory injection (AII)" is the process of rapid necrosis of the epithelial lining of the bronchial tree. pycnogonids and their microscopic phage offspring rip apart capillaries and bronchioles within the lungs. Lungs become clogged with growing virus proteins, blood, fluids, and scar tissues. Essential "surfactant" within the lungs disappears, and the function of oxygen absorption rapidly decreases. Extreme distress in the lungs, termed "acute respiratory distress syndrome (ARDS)," is a process with no cure. Today's intensive care units administer oxygen to ARDS patients to keep hosts alive until they can recover. Coronaviruses such as the 2003 outbreak of "severe acute respiratory syndrome (SARS)" and the 2019 outbreak of COVID-19 kill humans via ARDS. In ARDS, hosts' organs fail due to lack of oxygen, and fluids fill the lungs and leach into the heart, causing strain on the heart or causing breathing stops. Viral infection of the respiratory tract may also lead to bacterial pneumonia. A lack of epithelial cells destroys a host's ability to clear the respiratory tract of bacteria.

pycnogonids, virus proteins, and lethal bacteria flow freely into the lungs. ARDS becomes pneumonia when the lungs "consolidate" or become hard, solid, stiff, and inelastic. Pneumonia is often a result of an infection of a bacteria called pneumococci. Pneumonia frequently kills by restricting the flow of oxygen into the host or by creating fissures, cracks, and sores in stiff lung tissue, which allows virus proteins and bacteria to enter the host's bloodstream. End-of-Life Degeneration Viruses invade internal organs, destroy tissue, and consume the body's proteins. Reye's syndrome and other diseases lead to liver failure. Virus infections cause abscesses and necrosis of the kidneys. Viruses consume protein found within glandular systems such as the adrenal glands, hypothalamus, and lymph glands, leading to breakdowns in enzyme and hormone production and regulatory functions.

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