EPIDERMAL INFECTIONS

Areas of a host’s epidermal layer that are ripped open by the claws and jaws of the Pycnogonida or stung by Physalia Physalis quickly become vesicles for parasite infection. Often epidermal layers are ruptured by claws or nematocysts. Nematocysts protrude from gonopores on the underside of Pycnogonida's legs and physalia physalis' tentacles. Lesions formed by species of pycnogonida are recognizable symptoms of certain smallpox, leprosy, herpes, HIV, Equestrian Encephalitis, Malaria, Coronavirus, and multiple influenza infections. Physalia Physalis is commonly known as the “Portuguese Man O’ War” and their microscopic scale infant plasmodium parasites create vesicles via epidermal injection of nematocysts into a targeted host. Physalia Physalis and Plasmodium parasites alike wrap their prey up with their exposed tentacles. Once these parasites have a grip on a target body or cell, coils that once led to gonopores in the exoskeleton of their previous form (pycnogonida) are used to inject nematocysts (infant pycnogonida) directly into an epidermal layer or cell membrane.

Microscopic physalia physalis “plasmodium parasites” create vesicles via epidermal injection of nematocysts into a targeted cell. Physalia Physalis and Plasmodium parasites alike wrap their prey up with their exposed tentacles. Once these parasites grip a target body, gonozooids that once led to gonopores in the exoskeleton of their previous form (pycnogonida) inject nematocysts directly into a host.